Sworder

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  • Sworder
    Member
    Hormone changes, in general do it. Androgens do it too, increases the sebaceous glands output and gives you acne as well. There are so many things that can cause it you can’t simply look at E2. E2 should be in normal-low range anyway and he should supplement with an AI if it is high. Should be able to tell without tests. It would be silly to think that E2 alone results in acne, if so all the ladies would be covered in acne all the time.
    Sworder
    Member
    Genetics yes, but what does genetics tell us? It sends out hormones, we are altering genetics in a way by supplying hormones. This is why we can grow past "genetic potential", all these signals are going to be recorded one way or another from my perspective. If you are supposed to per say get 1.2grams of testosterone in a year naturally I believe you are maturing your endocrine system by exogenous supply of hormones.

    If you think about it, what does aging mean? It has to be the cumulative stress on the body by sex hormones. Genetics are the boss and uses hormones to send a message. I would think that it only makes sense that we are altering "genetics" by exogenous "messengers".

    The pax 7 genes are the ones responsible for myogenesis in the homeobox.

    @ Marcus3000 Your endo seems to talk a lot and be very general; care to try to squeeze some more information out of him about what he means with the statements. Specifics please!

    @Binsser What Matt stated is very true, it is known by people who have a clue and saying otherwise is probably "scare tactics".

    Sworder
    Member
    I usually always say after 10 days go to a doctor! Other than that I don’t feel the need to stress ’em out.

    I try not to call white Zebra when it’s just a horse

    Sworder
    Member
    Quote Originally Posted by asiandudeView
    Btw, have you found out how estro causes water retention? Or do you still think that water molecules pop out of the estrogen-receptor complex?

    I never said that they popped out of the ER complex. This is now a Sworder thread.

    I am logging off, if you want to contribute to the thread, so it’s not completely derailed. Please explain to everybody how and what causes the mandible to stop growing.

    Sworder
    Member
    I don’t use Google or Wikipedia.

    Reconstruction of the Pediatric Maxilla and Mandible
    http://archotol.jamanetwork.com/arti…ticleid=404258

    Mesenchymal in origin, bone is a living tissue making up the majority of the human skeleton. The craniofacial skeleton, including the mandible, grows through 2 mechanisms: epiphyseal proliferation and remodeling. Epiphyseal proliferation is largely responsible for increases in bone length and projection, a process that is dominant during the first 18 years of life. After age 18, the epiphyseal plate, located in the proximal zone of the conical subcondylar ridge, fuses. Prior to fusion, it exists as a 3-dimensional structure that, under the influence of the surrounding soft tissues, is essential to normal mandibular projection. The epiphysis adapts the intercondylar distance to the widening cartilaginous synchondrosis of the cranial base, highlighting the ever-important relationship between normal mandibular growth and normal basicranial development. The role of epiphyseal growth, particularly in the prepubescent pediatric patient, cannot be overemphasized.

    Sworder
    Member
    Creatine or aforementioned dose of testosterone .

    EQ is common in sports, but that’s a very long ester. You don’t want to use that right?..

    Sworder
    Member
    Quote Originally Posted by MuscleInkView
    Par for the course. Apply heat source and light massage or a warm soak in the tube. 400-800mg Motrin for pain.

    Watch for following symptoms:

    – fever
    – nausea
    – discoloration/redness
    – site remains hard
    – further swelling
    – loss of movement/range of motion

    These symptoms could indicate an infection and requires medical attention.

    What size of needle did you use? Are you rotating your injections? What are you injecting and dose?

    Am sure he will identify with over 50% of those symptoms

    Sworder
    Member
    Him coming on here asking about it means he knows that he needs to be sanitary which makes me think that he was super clean and used a new pin for each injection. He has clearly read about infections as he mentions "pinkish skin". I feel bad for these guys cuz they seem so stressed out. Infections are more prone to happen to the guy who has been around the block, who isn’t really careful anymore. Either way, he is having a chemical inflammation which is why it blossomed so quickly, infections usually take longer to develop and the muscle wouldn’t be stiff. After 10 days I would go to the doctor. Having PIP for a week is excessive but happens. 10 days is when I would think about it.

    These are the reasons why I dismiss it so quickly

    Sworder
    Member
    What are you trying to educate me in? Obviously you don’t know what you are talking about otherwise you wouldn’t question that bones in the mandible have growth plates. If the bones in the mandible doesn’t, how do they stop growing? The reason why I am not in the mood is because I don’t feel like correcting YOU and making this a SWORDER thread as usually is the case. Feel free to give some ambiguous post which make it seem like you were misinterpreted by me, but honestly it would be best if you didn’t post unless it entails me being wrong in something. Which is not the case unless you want to refute the data posted below. Most of the time I post or contradict AFTER reading literature about the subject; so before poking the beehive check your facts. I am done here, thanks I got work to do.

    Bone Development and Growth
    http://web.ardark_sideve.org/web/200807091…ne_growth.html

    Endochondral Ossification

    Endochondral ossification involves the replacement of hyaline cartilage with bony tissue. Most of the bones of the skeleton are formed in this manner. These bones are called endochondral bones. In this process, the future bones are first formed as hyaline cartilage models. During the third month after conception, the perichondrium that surrounds the hyaline cartilage "models" becomes infiltrated with blood vessels and osteoblasts and changes into a periosteum. The osteoblasts form a collar of compact bone around the diaphysis. At the same time, the cartilage in the center of the diaphysis begins to disintegrate. Osteoblasts penetrate the disintegrating cartilage and replace it with spongy bone. This forms a primary ossification center. Ossification continues from this center toward the ends of the bones. After spongy bone is formed in the diaphysis, osteoclasts break down the newly formed bone to open up the medullary cavity.

    The cartilage in the epiphyses continues to grow so the developing bone increases in length. Later, usually after birth, secondary ossification centers form in the epiphyses. Ossification in the epiphyses is similar to that in the diaphysis except that the spongy bone is retained instead of being broken down to form a medullary cavity. When secondary ossification is complete, the hyaline cartilage is totally replaced by bone except in two areas. A region of hyaline cartilage remains over the surface of the epiphysis as the articular cartilage and another area of cartilage remains between the epiphysis and diaphysis. This is the epiphyseal plate or growth region.
    Bone Growth

    Bones grow in length at the epiphyseal plate by a process that is similar to endochondral ossification. The cartilage in the region of the epiphyseal plate next to the epiphysis continues to grow by mitosis. The chondrocytes, in the region next to the diaphysis, age and degenerate. Osteoblasts move in and ossify the matrix to form bone. This process continues throughout dark_sideldhood and the adolescent years until the cartilage growth slows and finally stops. When cartilage growth ceases, usually in the early twenties, the epiphyseal plate completely ossifies so that only a thin epiphyseal line remains and the bones can no longer grow in length. Bone growth is under the influence of growth hormone from the anterior pituitary gland and sex hormones from the ovaries and testes.

    Even though bones stop growing in length in early adulthood, they can continue to increase in thickness or diameter throughout life in response to stress from increased muscle activity or to weight. The increase in diameter is called appositional growth. Osteoblasts in the periosteum form compact bone around the external bone surface. At the same time, osteoclasts in the endosteum break down bone on the internal bone surface, around the medullary cavity. These two processes together increase the diameter of the bone and, at the same time, keep the bone from becoming excessively heavy and bulky.

    Sworder
    Member
    Quote Originally Posted by asiandudeView
    do you know where are the epiphyseal plates located in the mandible?

    I am not in the mood, same place as every other bone in the body that grows.

    Sworder
    Member
    Quote Originally Posted by MickeyKnoxView
    It was 405.

    I guess Sworder and Phased have no idea about E2 and acne? I try to locate some information for you two to take a look at and bring yourselves up to speed.

    Cheers!

    LMAO how many pimples do u get / pg e2? Is it like a 2:1 ratio or ?

    Sworder
    Member
    I thought baseball was more about strength? Maybe your coach will place you on one of the bases in the outfield. Bulk up and start hitting the home-runs!

    500mg Test P/week go for mass! Like Barry Bonds

    Quote Originally Posted by WaterprimeView
    hmmm 500 mgs sounds a bit high thats more of bb dose but thanks anyway.

    Why are you using TrueMaxding drugs if you don’t want the same effects? 150mg/week would be stupid unless you are hypogonadal…

    Sworder
    Member
    Normal. Relax. if it persists past 10 days then you can see a doctor. But until then don’t even think about it, it’s normal!!
    Sworder
    Member
    Both GH and testosterone (androgens) are responsible for mandibular growth. If this is possible after the epiphyseal plates have closed, I wouldn’t think so.

    Sworder
    Member
    None the less steroids will aggravate your heart. It’s one of the most severe side effects of AAS use and nobody really think about it. All you hear is "I have no sides, no gyno, acne or night sweats" honestly, who cares about compared to your cardiac health?

Viewing 15 replies - 31 through 45 (of 57 total)