Xself

Haha, nice to see some familiar faces!

Thought I’d drop in,
been awhile.

I see the OP (great work btw) was worried about his nuts.
Well yeah, I’m going to share what I think, and what my latest andrologist said.

First the andrologist, and this is some time ago so new research might have emerged since.
But he also based this on his experience with patients, with was pretty extensive.

He said, and I’ve read the same study; that very high testosterone levels alone can sustain sperm production. We are then talking about extremely high levels, since ordinary, tour testes has about 30-100x the level of test that the rest of your body has.
Thus; dosages in excess of 1500mgs weekly was his approximation, which some guys do, but doing those dosages for any length of time? Your nuts isn’t the only thing you need to worry about.

But I think all that is pretty much guess work really, if I were to draw any conclusions from it I’d rather say; short heavy cycles (with test) are better for your nuts than more moderate dose long cycles.

Then there’s the genetics thing, and then there’s what I call “fucked up squared law”.
Genetics mean some people are not that fertile to begin with, they might need some treatment with hcg etc just to reproduce naturally.
(And mind you, reproducing naturally when 20yo, and 30’ish yo might be two very different scenarios)

Then the “fucked up squared law”;
It pretty much means; you’d be amazed how many people survive and recover from things you’d never think.

In short;

Regarding your nuts; it’s the duration, (and some compounds might be particularly bad like tren , but IMO; they’re just very potent at shutting you down), not the dose which matters.
And the duration is individual; while you’ll be shutdown quickly after any AAS dose of note, how quickly you bounce back is individual, and it’s faster when you’re young.

Last thing about that; not everyone gets 100% shutdown, at least in terms of sperm production. However, that you didn’t get shut down when you were 20, doesn’t mean you won’t when 30 or 40.
The only rule there is that everyone will get LESS fertile on cycle; but hey, if you’re still having some sperm alive, and doing it like rabbits, we all know plenty examples of that.

Hcg is your friend if your doing long cycles though. Or any cycle basically.

35mg? Sure their not 50mg? Lots of UGLs over here have begun pouring out 50mgs of DBOL and such.

I have to say it, I wouldn’t trust anavar to be doses at 35mg if it weren’t in a box that stated 35mg, even if my brother were the supplier.

I’ve gotten 50mgs Winstrol tabs that the supplier said;
"They’re more like 40mgs", which is another matter, cause he’s just admitting that he thinks they were underdosed. For me it wasn’t a problem if the pill was 30, 35, 40 or 50mg. But not a product I would give a woman.

And high fat diets are for Eskimos IMO.

Protein:30%, carbs 40%, fat 30% (of total energy intake)
is a simple enough maBamb set up I’ve felt good doing.
What carbs and fat you eat, and the timing of high GI food regarding fat intake matters too. Especially the first part.
10grams of trans fatty acids aren’t what you wanna mix in your protein shake.
10grams of MCT oil another matter.

And fructose, well it does help to mix that fructose with glucose, as it is in fruit.
But high fructose corn syrup or just much fructose is a problem;
Fructose skips the phospofructokinase-1/PFK-1 enzyme step in glycolysis, which happen to be the most important rate limiting step.
Then, fructose is mostly absorbed by the liver, unlike glucose which can be just as easily absorbed by the muscles directly.
Since the fructose that enters the liver has already committed itself to glycolytic breakdown it cause a rise in acetyl-CoA when it isn’t needed. The liver makes triglycerides to store this energy.
Short story: fructose can increase triglycerides in your blood, and be bad for your health.
(Some fructose is ok ofcourse, dont go nuts)
I may have forgot or confused some of the principles of fructose breakdown as i didn’t care to look it up,
Please correct me if it’s wrong, but the result is correct; increased triglycerides and other issues.

Ok, now that the math seem to be settled, let me fuck it all up by coming with this statement:
-are you sure anavar at those dosages doesn’t reach saturation of enzymes and therefore follow zero order kinetics?
That would mean the half life is no longer 9 hours.

Haha, had to say it.
You are overthinking this.

Anavar is pretty side effect free, but I’d choose TBOL over it any day.
Very gentle (for me), and actually really anabolic as well.
(Much more than var, more like DBOL actually)

Winny doesn’t cause appetite suppression in me, but a general feeling of being unwell/back ache, the depot doesn’t do this however.
(Some appetite suppression too, maybe)
But I’ve checked and my kidneys are fine on it, so I don’t know,
but I do know that stopping the compound around 14 days, and then start up again (pulsing) resolves it.

Anadrol can cause much more issues, appetite being one of them,
but either using it 3-4x a week or pulsing fixes that.
And I feel pulsing is way more effective than the 3x a week approach.
(Quite naturally as the total dose used will also be higher)

I feel that most orals kick in very fast, and I am after all on test (and maybe some other injectable), so spikes in concentration with orals isn’t something I worry about. Actually (though I have no proof of this) I think the spikes in blood concentration that orals give, increase IGF1 secretion more.
Whatever the reason, whether pulsing or even using 3x a week seem to help growth a lot. Another approach I’ve tried is alternating orals and dose,
like anadrol 3x a week at 50mg, 15-20mg DBOL between.
But this leads to unpleasant effects also, but it takes a while longer.
(Whether it’s just the dose reduction or if alternating the compounds also play a role I’m not sure, though i think that depends on the oral. DBOL and TBOL treat me real nice, drol and winny doesn’t in the long run, not to mention halo.)

But why complicate? Pulsing works and is easy.

This is pretty much the same as the discussions over timing of Dbol .
As most of you know, methandrostenolone is listed as having a very short half life, sometimes down to just 3 hours.
However, when it comes to real life reports,
Many say that a single dose of 50mg works just as well as the same amount split throughout the day.
(This might be different if running dbol only though, as I guess most people would stack it with an injectable, which changes the game somewhat)

Personally, if I took an oral only with a half life less than 10 hours like anavar ,
I’d take the highest dose in the morning, and save a smaller dose for later.
My thinking is this:
Say I take 60mg in the morning, then (if we shall follow straight half life kinetics) I’d have a little less than 30mg in my system 12 hours later.
Taking another 20mg at this point would kick that up to nearly 50mg in your system, leaving somewhat less than 25mg when it’s time for the next 60mg intake.

Why take more in the morning or at one time?
The higher the dose, the higher IGF1 response, among other things.