xzed

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  • xzed
    Member
    Quote Originally Posted by marcus300View
    Hell man just look at their screen names. That will tell you the caliber of people we are dealing with.

    Is this your way to understand? Perfect

    xzed
    Member
    Quote Originally Posted by Mr.BBView
    To kick start the gonads.

    Hypogonadism can be primary or secundary, primary is directly related to the testis, secundary can have a variety of reasons.

    So many ppl have already tried to tell you that HCG is used on cycle to maintain some testicular function so that when its time to restart the testis it will be easier. But of course this is not 100% sure like everything in medicine.
    It seems you don’t want to understand…

    I got the idea. Keeping own production. Better recovery…but I already know what you talked about. The problem is I couldnt maintain any testiculer function during cycle. I am trying to understand what was my wrong. I used pregnyl which come from farmacy and i was carefull about preparing and keeping etc. My testicles was got pretty hard atrophy and my blood levels were same after hcg . You guys really got better recovery with hcg?so prove it. I mean not with subjective values. Show that with blood levels, usg proved testicle size etc.
    And I dont understand why u all acting like hcg after cycle is big mistake. I saw a lot of doctor use it for treatment of eksojen testo induced hypogonotropic hypoginadism which u also can find on pubmed articles. After a good hcg treatment lh wasn’t supressed at all. It recover itself as the same speed of without hcg. My only worries about desensitized receptors which could come with high dosage.
    By the way hypogonotropic hypoginadism already mean seconder.

    xzed
    Member
    Quote Originally Posted by DomusView
    hCG is suppressive! Since we know that hCG mimics LH, then we know that in the presence of exogenous LH, the pituitary gland will not produce LH.

    Where this information come from? As I know LH can be suppressed by only androjens and can increase by gnrh which come from hypothalamus. Is there any negative feedback system between gnrh and lh?
    And if your information is right why damn company says use this drug for hipogonothropic patients. Why urologist dont let petients to produce their "natural" Lh if it could supresed by hcg ?
    By the way sorry for my first information. I didnt try to say hcg can increase lh. I just want to say if hcg works on cycle if leyding cells produce testo at least we have to see an increase on blood.
    On article which marcus shared there was an increase by 500ui/w hcg on 200mg/w testo cycle. And I am wondering now if 500 hcg ui/w wasnt not enough on my 500 mg testo cycle. Maybe this why I couldnt see testo increase. And another thing is if it works on cycle it doenst mean you cant use it post cycle which make more sense

    xzed
    Member
    Quote Originally Posted by renepjdView
    No clue what that means….

    I mean I am not woried about why doesnt lh increase. If its lh mimetic and if it increase own production at least I have to see increased Testo levels

    xzed
    Member
    Quote Originally Posted by renepjdView
    xzed you’re missing the point completely. HCG will not stimulate LH production, it only mimics it and will not show as increased LH on blood work.

    Doesnt matter. At least I was expecting to see increased testo levels if there was own production. I dont care lh

    xzed
    Member
    Quote Originally Posted by marcus300View
    HCG has the ability to maintain testicular function and size which means it will maintain endogenous testosterone production by stimulating the testes even when shutdown from using steroids has occurred.

    HCG is VERY important in cycles IMHO. It prevents the main reason the HPTA doesn’t recover immediately post cycle. If you maintain testicular function and size recovery is far better which is of great benefit to us all.

    Check out this study it will help you understand what its capable of doing

    ps, how old are you?

    24 years old.
    Great article. I can read that the serum test levels increased after hcg even on testo. Mine wasnt same. I don’t know maybe thats because of I used 500 mg per week also some other anabolics with. In this article they use 200 mg which could let to hcg to effect to leyding cells and which is not enough for TrueMaxding.
    I am not sure if Intratesricular testosteron is good indicator for own testesteron production. And I don’t get why they don’t check blood testo levels after exojen testo eliminated from blood. It could show own production better then intratesticular testesteron

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