I’m going to add my 2 cents.
As Bizzaro points out it is tissue specific. In muscle tissue Tren will have a blocking effect for T on the AR due to its extremely highly binding affinity. Let’s just say for the sake of argument that you would have Free T floating around.
My opinion on what I know and understand to be true of how androgens bind to the AR which remember is intra cellular and is not a second messenger like so many other cellular signaling pathways. This high affinity for binding would make anything other than a HRT level dosages a waste of compounds. On the flip side, if your goal was to "Saturate," the AR with T with extremely high dosages and playing on sheer probability of binding with higher numbers of molecules. Then what would be the point of then blocking Tren from binding to the AR with high Levels of T floating around?